Researchers genetically restored levels of the SynGAP protein in adult mice who were deficient. Restoring the protein levels resulted in improved memory and a reduction in seizures related to the SYNGAP1 disorder, a form of autism. Findings suggest it is possible to extend the window of treatment for those with this specific form of ASD, beyond infancy and into adulthood.
from Neuroscience News Updates http://bit.ly/2XRdYXn
from Neuroscience News Updates http://bit.ly/2XRdYXn
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